Aad goudriaan biography of donald
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High Court rules sacked automotive firm boss is entitled to €1.5m shares
It was also claimed a failure to address important advice given by financial advisers, Rothschild, and a failure to appraise shareholders, meant it had to face a crisis in 2016 which could have been mitigated if Mr Goudriaan had acted earlier.
He denied those claims and said after joining the group in 2010, he learned it was not as financially strong as he was led to believe.
Today, Mr Justice Michael Quinn ruled Mr Goudriaan was entitled to a decree that the Dublin-registered shareholding firm, Jenda Corporate Holdings, along with BosalNetherlands BV and a linked company, United Trustees (NZ) Ltd, must specifically perform an element of his (Goudriaan's) contract that he is entitled to acquire 155 shares in Bosal worth €1.5m.
The defendants claimed the period during which he was entitled to exercise that share option plan had expired.
Mr Justice Quinn said the service agreement Mr Goudriaan entered into with the company, and under which he could exercise the share option, could only be terminated on 15 days' notice.
The judge said Mr Goudriaan was initially offered "garden leave" which he refused. On October 28, 2016, Jenda, as sole shareholder of Bosal, passed a resolution suspending him "with i
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ARMO Original Products Showc • Published in final edited form as: Curr Top Behav Neurosci. 2022;54:245–281. doi: 10.1007/7854_2021_273 The urge to seek and consume excessive alcohol is intensified by prior experiences with social stress, and this cascade can be modeled under systematically controlled laboratory conditions in rodents and non-human primates. Adaptive coping with intermittent episodes of social defeat stress often transitions to maladaptive responses to traumatic continuous stress, and alcohol consumption may become part of coping responses. At the circuit level, the neural pathways subserving stress coping intersect with those for alcohol consumption. Increasingly discrete regions and connections within the prefrontal cortex, the ventral and dorsal striatum, thalamic and hypothalamic nuclei, tegmental areas as well as brain stem structures begin to be identified as critical for reacting to and coping with social stress while seeking and consuming alcohol. Several candidate molecules that modulate signals within these neural connections have been targeted in order to reduce excessive drinking and relapse. In spite of some early clinical failures, neuropeptides such as CRF, opioids, or oxytocin continue to be examined for their ro Abstract